Metabolic Alterations associated with T2D are very well characterised by epidemiological and research based reports. The pathogenesis and development of T2D is ascribed to four mechanisms; improved Superior glycation stop products (AGE) development, improved polyol pathway flux, activation of protein kinase C (PKC) isoforms, and amplified hexosamine pathway flux[7]. Till https://www.directivepublications.org/journal-of-diabetology-research/